Gout cures come in many forms.
There are treatments to lower uric acid and treatments to relieve gout pain. There are gout medicines, and alternative remedies. There are established therapies, new therapies, and therapies in development.
Whilst reorganizing the Gout Treatment section, and other tidying up tasks, I’m reminded of another form of gout treatment – just stop whatever is causing your gout.
Now, it is not always possible to determine all causes of gout, and I explain some of the difficulties in the Gout Symptoms Section. However, I’ve just re-discovered an interesting report that might give you extra clues, if you take medication for other health conditions.
Diuretics are a well known cause of gout. They are often prescribed for high blood pressure, for which there are often alternatives that do not cause gout.
Hyperuricemia and gout: Some medications can ‘precipitate’ gout (Drug Induced Hyperuricemia & Gout), is a study by Norma Pearson, published in the Canadian Pharmacists Journal. It lists several potential gout-causing drugs, including:
|Increased purine ingestion||Pancreatic enzymes (pancreatin, pancrelipase)|
|Increased uric acid production (cell lysis, catabolic effect, increased WBC production, hemolysis)||Cytotoxic chemotherapy (e.g.,aldesleukin, asparaginase, busulfan, carboplatin, chlorambucil, cisplatin, cyclophosphamide, cytarabine, daunorubicin, fludarabine, hydroxy-urea, mechlorethamine, melphalan, mercaptopurine, thioguanine, vinblastine,vincristine), didanosine, ethanol, filgrastim, fructose, glucocorticoids, ribavirin/interferon|
|Reduced renal clearance of urate||ACE inhibitors (e.g.,lisinopril, ramipril, trandolapril), cyclosporin, diazoxide, diuretics (i.e.,acetazolamide, bumetanide, chlorthalidone, ethacrynic acid, furosemide, indapamide, metolazone, thiazides, triamterene), ethambutol, levodopa, pyrazinamide, salicylates (low dose), tacrolimus|
|Increased urate production & decreased clearance||Niacin|
The report lists several other drugs, some of which might have alternatives. Even where there are no alternatives, if the medicine at fault is not a permanent requirement, it is good to know that you might be able to stop gout treatments when the other condition is cured.
Whatever your situation, it is always wise to discuss potential side-effects and drug interactions with your doctor or pharmacist. As the report concludes:
Gout has been linked to many medications (see Table 3 [additional to table above]). By identifying patients who are at higher risk for hyperuricemia, pharmacists can monitor patients when suspected agents are added to their regimen.
Before deciding which is the best gout treatment for you, see if you can remove the cause first. Your best gout cure might just be the one you do not need.
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